The induction of metabolic alkalosis by correction of potassium deficiency.
نویسندگان
چکیده
منابع مشابه
Cardiac Necrosis Accompanying Potassium Deficiency and Administration of Corticosteroids.
• Cardiac necrosis was first recognized in conjunction with potassium deficiency by Schrader et al. in 1937. No serum or tissue analysis was done in this study. Follis et al. in 1942 found low heart potassium in rats that developed cardiac necrosis while being fed diets deficient in potassium. The occurrence of cardiac lesions in rats secondary to administration of desoxyeorticosterone or low p...
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We have demonstrated previously that deoxycorticosterone acetate (DOCA)/salt induces cardiac hypertrophy and left ventricular dysfunction independent of blood pressure (BP) in 1-renin gene mice. Because these mice also develop hypokalemia and metabolic alkalosis caused by mineralocorticoid excess, we investigated whether correcting hypokalemia by dietary potassium supplementation would prevent ...
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HAGGIE, S.J., FERMONT, D.C. & WYLLIE, J.H. (1976) Treatment of duodenal ulcer with cimetidine. Lancet, i, 983. HARKEN, A.H., GABEL, R.A., FENEL, V. & MOORE, F.D. (1975). Hydrochloric acid in the correction of metabolic alkalosis. Archives of Surgery, 110, 819. HERTZ, P. & RICHARDSON, J.A. (1972) Arginine-induced hyperkalemia in renal failure patients. Archives of Internal Medicine, 130, 778. KA...
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EDITORIAL SYNOPSIS Observations are described in an 8-year-old boy, the fifth case to be recognized of the syndrome previously known as 'congenital alkalosis with diarrhoea'. The condition appears to result from an abnormal handling of chloride ion in the alimentary tract; the pathognomic biochemical features are an invariably high faecal concentration of chloride and liquid stools which consis...
متن کاملGitelman syndrome combined with complete growth hormone deficiency
Gitelman syndrome is a rare autosomal recessive hereditary salt-losing tubulopathy, that manifests as hypokalemic metabolic alkalosis, hypomagnesemia, and hypocalciuria. It is caused by mutations in the solute carrier family 12(sodium/chloride transporters), member 3 (SLC12A3) gene encoding the thiazide-sensitive sodium chloride cotransporter channel (NCCT) in the distal convoluted tubule of th...
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عنوان ژورنال:
- The Journal of clinical investigation
دوره 45 4 شماره
صفحات -
تاریخ انتشار 1966